2 edition of Effects of a sucrose-rich diet on rats and humans. found in the catalog.
Effects of a sucrose-rich diet on rats and humans.
S. J. Forrestal
Written in English
|The Physical Object|
|Pagination||39 leaves, ap.p.endices|
|Number of Pages||39|
Cacao extract enriched in polyphenols prevents endocrine-metabolic disturbances in a rat model of prediabetes triggered by a sucrose rich diet Author links open overlay panel María Cecilia Castro a 1 Hernán Villagarcía a 1 Ada Nazar a Luisa González Arbeláez b María Laura Massa a Héctor Del Zotto a José Luis Ríos c Guillermo R. Overconsumption of sugar-sweetened beverages, in particular carbonated soft drinks, promotes the development of overweight and obesity and is associated with metabolic disturbances, including intrahepatic fat accumulation and metabolic syndrome. One theory proposes that drinks sweetened with high-fructose corn syrup are particularly detrimental to health, as they contain fructose Cited by:
In this work, we studied the effect of a short-term (3 wk) and a long-term (15 wk) administration of a sucrose-rich diet (SRD) to Wistar rats on the morphological aspects and metabolic function of Cited by: We examine the effects of feeding a high-sucrose diet on body weight gain, plasma triglycerides, and stress tolerance in rats. Feeding a high-sucrose (60%) diet for 2 weeks did not induce a.
The Interaction of Diet and Stress in Rats: High-Energy Food and Sucrose Treatment Nancy K. Dess and Seung Choe Occidental College Thomas R. Minor University of California, Los Angeles Exposure to inescapable shock typically reduces eating and body weight in rats. The present study examined the modulation of stress effects by prestress diet and. A high sucrose diet may induce alterations in calcium balance in humans (Ericsson et al. , Lemann et al. , Lindeman et al. , Thom et al. ) and in bone composition in animals (Li et al. , Saffar et al. , Saffar and Makris , Salem et al. , de Tessiers and Saffar ).Our previous studies on the effect of a high sucrose diet demonstrated that the diet itself.
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Therefore OP rats still consumed more total sucrose (liquid and diet combined) than OR rats during h two bottle testing, perhaps indicating a reduction in the ability to detect luminal sugars.
However, during the OGTT, HE-fed OP rats had a better response to the glucose load than OR rats, indicating that OP rats do not exhibit deficits in Cited by: At autopsy, liver nodules were observed in the dead animals, but the cause of death was not clear.
After the diet was changed to a sucrose-rich diet in which 50% of the starch was replaced by sucrose, no deaths occurred in the animals during the period of : Akika Sakamoto, Michishi Goya, Yoko Degawa, Masayuki Mitsui, Toshio Mori, Kazutoshi Tamura, Masahiro.
The duration of intermittent access to preferred sucrose-rich food affects binge-like intake, fat accumulation, and fasting glucose in male ratsCited by: 2. The N-acetylcysteine-induced reduction in intestinal absorption of the ingested sucrose may be contributed for the slimming effect it provokes in rats subjected to the sucrose-rich diet.
It is well known that the absorption and delivery of dietary compounds by intestinal cells are part of a complex process, which is influenced by the physiological state of by: EFFECTS OF SUCROSE-RICH DIET IN ENDOCRINE PANCREAS challenge, in normal Wistar rats fed a moderate amount of fructose (15% to 20%) for 7 months.
However, they recorded no differences either in body weight or in relative food intake as compared with age-matched by: A sucrose-rich diet, due to its high content of fructose, induces in the rat: glucose intolerance, associated with hyperinsulinemia, increased plasma free fatty acids (FFA) and hypertriglyceridemia, changes similar to the ones found in human diabetes type 2 also called non-insulin-dependent diabetes mellitus (NIDDM) and attributed to insulin resistance.Cited by: 9.
Rats in the Sucrose group had free access to 10% sucrose solution during the day diet intervention and during gestation, with access ceasing at parturition (P). Dietary constituents, such as sucrose, may influence renal integrity during aging.
Diets with high sucrose content cause extensive basement membrane damage in retinal vessels of WKY rats (J Clin Lab Sci), which have been likened to the changes in diabetes mellitus and aging.
Nevertheless, it is unknown whether similar lesions also occur in renal glomeruli and play a role in the Cited by: 3. diet-induced myocardial remodelling in rats. Curr Res Cardiol ;2(1) BACKGROUND: A high-carbohydrate diet leads to the metabolic syn-drome (MetS), which leads to an increased risk for cardiovascular dysfunc-tion; however, the effect of high-carbohydrate diets on systemic metabolism has not yet been fully by: 1.
The present study characterizes effect of a high-sucrose diet on FGF21 production and related metabolic parameters.
To compare the effects of sucrose on FGF21 production, mice were fed a high sucrose or a high-starch diet containing identical amounts of protein Cited by: Jordy, A.
et al. Insulin sensitivity is independent of lipid binding protein trafficking at the plasma membrane in human skeletal muscle: effect of a 3-day, high-fat diet.
Physiol. Regul Cited by: Metabolic and behavioral effects of a high-sucrose diet during weight loss. Metabolic and behavioral effects of a highsucrose diet.
feeding in rats (17). In humans, sucrose has been shown to. One is table sugar — sucrose. The other is high-fructose corn syrup. High-fructose corn syrup has become ubiquitous in soft drinks and many other processed foods.
Lustig presented his case against fructose in a recent UCSF Mini Medical School course on diet and nutrition, part of a series sponsored by the Osher Lifelong Learning Institute. There’s a ton of incredibly promising intermittent fasting (IF) research done on fat rats. They lose weight, their blood pressure, cholesterol, and blood sugars improve but they’re rats.
Studies in humans, almost across the board, have shown that IF is safe and incredibly effective, but really no more effective than any other diet. Sucrose-enriched diet-fed rats showed a decrease in food, lipids and protein intake as well as in serum total cholesterol levels, an increase in carbohydrates intake, glucose, insulin.
Request PDF | Dietary fatty acids effects on sucrose-induced cardiovascular syndrome in rats | Cardiovascular disease is one of the leading causes of morbidity and mortality in Mexico. The inflammatory profile and liver damage of a sucrose-rich diet in mice indicating beneficial effects for a sucrose-restricted diet.
Animals and humans fed high-energy diets based on fructose or sucrose exhibit an elevation in cholesterol due to an imbalance between the production and clearance of very low-density lipoprotein cholesterol Cited by: Abstract.
Energy intakes, body weights, body fat index and intrascapular brown adipose tissue (IBAT) were examined in adult male Hooded-Wistar rats given a sucrose solution drinking option and one of five solid diets for 33 days: 1) a 12% fat, starch diet; 2) a 12% fat, sucrose-starch diet; 3) a 40% fat, starch diet; 4) a 60% fat, starch diet; or 5) an 80% fat by: Effects of diets rich in sucrose, coconut fat and safflowerseed oil on the development of the obese hyperglycaemic (ob/ob) syndrome in mice February Diabetes research (Edinburgh, Scotland) The insulin-like effects of vanadate were compared in streptozotocin-induced diabetic rats fed on high starch control and high sucrose diets for a period of six weeks.
Diabetic rats in both diet groups were characterized by hypoinsulinemia, hyperglycemia (– fold increase) and significant decreases (p.
In summary, in contrast to the substantial effects on offspring of maternal diets that contain 65% sucrose ; 50% fructose or 50% sucrose ; sweetened-condensed milk or 20% sucrose solution, in the present study feeding female rats 10% sucrose produced only limited and subtle effects.
This was despite the females’ development of glucose intolerance and hypertriglyceridemia after four weeks of sucrose Cited by: In our study, free access to the sucrose-rich diet and high food consumption caused obesity/abdominal obesity in the EG rats from week Obesity was associated with increased serum levels of glucose, triglycerides, VLDL-cholesterol, and insulin, which are manifestations of insulin resistance [9, 49].
The hyperinsulinemia led to increased hepatic synthesis of fatty acids, triglyceride accumulation Cited by: The effect of saccharin intake on the consumption of sucrose was investigated. Groups of rats were offered various combinations of rat stock diet, stock diet + % Na-saccharin, granulated sucrose, sucrose in water and saccharin in by: